Gonadotropin releasing hormone antagonist drugs

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Actual: Gonadotropins, gonadotropin-releasing hormone agonists, and gonadotropin-releasing hormone antagonists are gold-standard drugs in ovarian stimulation to increase the number of follicles and to prevent spontaneous ovulation. The field remains in a continuous evolution towards personalized medicine.
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In men, gonadotropin-releasing hormone antagonists inhibit the release of luteinizing hormone, and consequently less testosterone is produced. Testosterone stimulates the growth of many forms of prostate cancer. Gonadotropin-releasing hormone antagonists are used to treat prostate cancer as by reducing the levels of testosterone the size of

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Uterine Fibroid Treatment Drugs Market - By Drug Class (Progestin-releasing Intrauterine Device, Contraceptive, Gonadotropin-releasing Hormone Antagonist)

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Drug Insight: clinical use of agonists and antagonists of luteinizing-hormone-releasing luteinizing-hormone-releasing hormone (LHRH), also known as gonadotropin-releasing hormone. In

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Gonadotropin-releasing hormone (GnRH) antagonists are synthetic analogs of gonadotropin-releasing hormone, which is produced by the hypothalamus and controls the secretion of follicle stimulating hormone (FSH) and luteinizing hormone (LH) by the anterior pituitary.

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List of Gonadotropin-releasing hormone antagonists. View by Brand

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A gonadotropin-releasing hormone antagonist (GnRH antagonist) is a synthetic peptide that competes with the neurohormone GnRH for its

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Gonadotropin-releasing hormone antagonist (GnRH antagonist), such as Antagon and Cetrotide. These drugs aren't pills that you swallow.

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Gonadotropin-releasing hormone antagonists (GnRH antagonists) are a class of medications that antagonize the gonadotropin-releasing hormone receptor (GnRH

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Gonadotropin-releasing hormone receptor antagonists, as selective gonadotropin-releasing hormone receptor blockers, reduce the release of follicle-stimulating hormone and luteinising hormone from the pituitary, resulting in anovulation and amenorrhea in a dose-dependent manner.1 The suppression of gonadotropin-releasing hormone receptor presents several potential therapeutic options for

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